Klüver-Bucy syndrome is a rare neurological disorder that may occur as a transient or permanent syndrome[^1].
Historical
Klüver-Bucy syndrome was first described in 1937 as an experimental neuropsychiatric syndrome in monkeys with bitemporal brain lesions and was named after the German-American neuropsychologist Heinrich Klüver (1897–1979) and the American neurologist and neurosurgeon Paul Bucy (1904–1992)[^1].
Symptoms
The most common manifestations of the syndrome include hyperorality with altered eating habits, hypersexuality, and visual agnosia. Seizures, hypermetamorphosis, and amnesia may also occur with increased frequency[^1].
Etiology
Klüver-Bucy syndrome may occur in patients with bilateral lesions of the Temporal lobe, including the Hippocampus and the Amygdala. Klüver-Bucy syndrome may occur in the context of various neurodegenerative, traumatic, nontraumatic, and infectious brain insults[^1].