Klüver-Bucy syndrome is a rare neurological disorder that can occur as a transient or permanent syndrome[^1].
Historical
Klüver-Bucy syndrome was first described in 1937 as an experimental neuropsychiatric syndrome in monkeys with bitemporal brain lesions and was named after the German-American neuropsychologist Heinrich Klüver (1897–1979) and the American neurologist and neurosurgeon Paul Bucy (1904–1992)[^1].
Symptoms
The most common manifestations of the syndrome include hyperorality with altered eating habits, hypersexuality, and visual agnosia. Seizures, hypermetamorphosis, and amnesia may also frequently occur[^1].
Etiology
Klüver-Bucy syndrome can occur in patients with bilateral lesions of the temporal lobe including the hippocampus and the amygdala. Klüver-Bucy syndrome may occur in the context of various neurodegenerative, traumatic, non-traumatic, and infectious brain conditions[^1].